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Aging further exacerbates vulnerability: in aged or overweight mice, reduced basal NRF2 antioxidant capacity and diminished mitochondrial reserve in aged or overweight mice lead to exaggerated ROS surges and excessive UPR activation during I/R (120), whereas short-term fasting reinduces NRF2, lessens acute necrosis, and mitigates BUN elevation in these models, consistent with metabolic reprogramming as a protective mechanism

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Acetylcholinesterase activity in lipopolysaccharides-induced neuroinflammation rat brains was reduced by honey

It plays an important role in brain function, DNA synthesis, and red blood cell production

For instance, experimental studies that involve glutathione (Yamasoba et al., Figure 2 Consistent with this idea, glutathione, a tripeptide molecule (cysteine, glutamic acid, and glycine) and the most abundant endogenous free radical scavenger in humans, was the first antioxidant evaluated for the treatment of NIHL (Yamasoba et al., N-acetylcysteine (NAC), a cysteine precursor, is an antioxidant molecule that enhances the production of glutathione (Kopke et al., a replenisher of this endogenous scavenger (Kopke et al., Interestingly, recent evidence suggests that HK-2, a multifunctional antioxidant with a metal chelator and free radical scavenger properties (Kawada and Kador, Several micronutrients such as vitamins A, C, and E show antioxidant properties and have been used either individually or combined as experimental treatments for NIHL

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