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The pathophysiology of sepsis is underpinned by a complex interplay of immune activation, endothelial dysfunction, and profound metabolic and mitochondrial dysregulation

Figure 1 2.7 Calculations and statistics All data were statistically analyzed using the MIXED procedure of SAS version 9.4 (SAS Institute Inc., Cary, North Carolina, USA) with the restricted maximum likelihood method

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Activated cells synthesize and release a variety of cytokines and inflammatory mediators, as well as activate signaling pathways such as NF-B (Hailman et al., 1994), MAPK (Chang et al., 2019), toll-like receptors (TLRs) and STAT-1, which triggers the synthesis and release of cytokines and inflammatory mediators like interleukin-1 (IL-1), interleukin-6 (IL-6), tumor-necrosis factor- (TNF-

Symptomatic atherosclerosis is associated with an altered gut metagenome