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FIGURE 10 Notably, activators of SIRT1, such as metformin, have demonstrated the ability to inhibit Th17 cell differentiation and downregulate IL-17A and RORt levels by promoting STAT3 deacetylation (Zou et al., 2020), which ultimately suppresses the induction of RORt transcription ( In summary, our study primarily investigated the beneficial effects of GHK-Cu in UC, using a DSS-induced colitis mouse model and an in vitro co-culture system, suggesting that GHK-Cu may exert anti-inflammatory properties and its role in facilitating the repair of UC by regulating SIRT1/STAT3 signaling pathway

Mehrabani S, Khorvash F, Heidari Z et al (2023) The effects of synbiotic supplementation on oxidative stress markers, mental status, and quality of life in patients with Parkinsons disease: a double-blind, placebo-controlled, randomized controlled trial

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Retrieved May 12, 2013