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Piatti PM, Monti LD, Valsecchi G, Magni F, Setola E, Marchesi F, Galli-Kienle M, Pozza G, Alberti KG

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C5a, the anaphylatoxin produced following complement activation, is known to mediate potent nociceptive effects mainly by interacting with the G-protein coupled receptor (GPCR) complement component 5a receptor 1 (C5aR1) [12], and the activation of the C5a-C5aR1 axis has been associated with neuropathic pain [13] and neuro-inflammatory injury [14]

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Once your thyroid releases thyroxine (T4) into your bloodstream, certain cells in your body transform it into triiodothyronine (T3) through a process called de-iodination

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Proliferating cells thus require access to free cysteine to deal with oxidative stress

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A meta-analysis on the efficacy and safety of combined vitamin C and E supplementation in preeclamptic women

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After inducing ischemic AKI in 810-week-old, male C57BL/6 mice, mass spectrometry metabolomics revealed that the liver had the most distinct phenotype 24 h after AKI versus 4 h and 7 days

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