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In the APP/PS1 mouse model of AD, administration of AKG or calcium -ketoglutarate (Ca-AKG) ameliorated impaired long-term potentiation at hippocampal CA1 synapses, with more pronounced effects observed in female AD mice

Rathore J

[5] During the reaction, an amino acid residue in the enzymes active site acts as a general base and abstracts a proton from C-3, leading to the formation of the cis-2,3-enediol intermediate

What If the Reconstituted Solution Was Left at Room Temperature Overnight