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Metabolite concentrations for each sample were collected in data matrices

Oxaliplatin induces hyperexcitability at motor and autonomic neuromuscular junctions through effects on voltage-gated sodium channels

However, the mechanisms leading to endothelial damage remain to be clarified, prior to obtain promising new treatment strategies and to understand whether the endothelial injury is primarily involved in the pathology or reflects the consequences of COPD occurrence

Mutations in bone morphogenetic protein type II receptor cause dysregulation of Id gene expression in pulmonary artery smooth muscle cells: implications for familial pulmonary arterial hypertension

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Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol