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Similar observations were made in MDA-T32 and MDA-T68 PTC cell lines (Shown in Supplemental Fig

Neuropathological investigations in the substantia nigra pars compacta of Parkinsons disease patients demonstrate that deficient PGC-1 expression compromises mitochondrial biogenesis and disrupts the coordinated transcriptional activation of antioxidant stress-responsive gene networks, thereby precipitating dopaminergic neuronal degeneration (Piccinin et al., 2021)

However non-catalysed insertion is negligible when Co(II) is buffered to an availability approximating the mid-point of the dynamic range for RcnR [93]

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