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Lipid bilayer stress activates the unfolded protein response (UPR) via ATF6, IRE1, and PERK, with IRE1 modulating autophagy and lipolysis under lipid perturbation (123)

One region is termed as G-site, which is hydrophilic in nature and binds the physiological substrate GSH, and another region is the H-site, which assists in binding of electrophilic substrates by providing a hydrophobic environment (Frova, 2003)

M.HurtadoO.RomeraV

Funding This research was funded by the Qatar Foundation, grant number QB14 (PJT) and Qatar University, grant number QU ERG-CMED-2020-1 (NR)

This approach examines cell-specific Cre expression and activity