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Nuclear exclusion of p53 reportedly leads to translocation of p53 to mitochondria and transcription-independent induction of apoptosis [28], which may explain the therapeutic effect of FOXO4-DRI

In human cells, PGRMC1 deletion reduces CYP51A1 enzymatic activity, thereby suppressing cholesterol biosynthesis and promoting the accumulation of toxic sterol intermediates

Activation of mTORC1 elevates and activates SREBP1 and SREBP2 by multiple mechanisms 49 , and transcription of the gene encoding G6PDH is elevated by SREBP1 in an mTORC1-dependent manner 48

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When administered intravenously, vitamin C can reach blood concentrations up to 70 times higher than oral supplementation, creating powerful therapeutic effects