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While the primary mechanisms of CPZ toxicity are poorly understood, in vitro studies report CPZ toxicity to be limited to primary oligodendrocytes (Bnardais et al., 2013) while others suggest the requirement of pro-inflammatory cytokines (Pasquini et al., 2007)

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(b) a transcellular pathway, mainly across the sustentacular cells, most likely by receptor-mediated endocytosis, fluid-phase endocytosis, or passive diffusion (not for peptides)

Understanding the root cause is essential for effective treatment and long-term management

Extending the scope of research beyond localized tissue repair to organismal homeostasis