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However, GSH is naturally unstable and not very bioavailable orally, as it is rapidly degraded during digestion

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To counteract oxidative stress, therapeutic interventions have shown promise

CQ-mediated autophagy inhibition in JS-1 cells concomitantly attenuated ferritinophagy-associated signatures, as evidenced by diminished LC3B II expression, accumulated P62 protein, downregulated NCOA4 mRNA, and elevated FTH and FTL