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The ferroptosis-inducing drug, erastin, induces ferroptosis through the inhibition of the cystine/glutamate antiporter system xC-/xCT, which is essential for the synthesis of reduced glutathione, ultimately leading to the accumulation of toxic lipid peroxides (16)

The downregulation of SOD and upregulation of genes like MPO and HIF1 support this oxidative stress-driven mechanism

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This posttranslational modification, termed S -nitrosylation or S -nitrosation, requires either metals or O 2 to catalyze the formation of the nitrosothiol adduct

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