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doi: 10.1073/pnas.1717520115

For the volumes used in peptide research (typically 0.05 to 0.5 mL), this is rarely an issue

Taken together, the action mechanisms of AFB1-exposure-induced mitochondrial dysfunction may include direct damage to the mitochondrial ultrastructure, a decrease in mitochondrial membrane potential, inhibition of TCA cycle-rate-limiting enzymes and mitochondrial biogenesis, loss of ETC complex activities, and a decrease in ATP levels

In this study, we describe ROS, the ROS generation pathway in vivo , oxidative stress, and the harmful effects of oxidative stress on physiological functions

Enteral Nutr

[DOI] [PMC free article] [PubMed] [Google Scholar] 38.Thornton DJ, Sheehan JK
