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However, p53 3KR as an acetylation-deficient mutant of the p53 protein fails to induce cell cycle arrest, senescence, and apoptosis, which completely retains the ability to regulate SLC7A11 expression and induce ferroptosis in tumor cells under oxidative stress

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Horowitz, A

J., Stern, R

SCI leads to ROS accumulation in skeletal muscle [40], resulting in oxidative injury, a key contributing factor to skeletal muscle atrophy [41]

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