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10.1038/s41419-020-03194-2 240 XiaoR.WangS.GuoJ.LiuS.DingA.WangG.et al (2022)

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These changes in SOD2 expression are believed to contribute to the development and progression of DR, particularly by enhancing oxidative stress, mitochondrial dysfunction, and inflammatory responses in retinal tissues (112)

The mechanism of action of DSePA against RP involved its ability to suppress the radiation-induced sustained inflammatory response by upregulating the pulmonary activity level of GPx and concurrently blocking the NF-kB/IL-17/G-CSF/neutrophil axis (Table 2) (Fig
