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Approximately one-half of the 102 patients studied had Child's A, namely clinically compensated cirrhosis, although the other half included in the study had greater degrees of hepatic decompensation

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found the viability of the isoproterenol and IBMX-treated, RV-infected cells was significantly higher at later time points post infection as compared to non-treated RV-infected cells, suggesting that the reduction in RV-induced cytopathicity is correlated with increased cell viability (Cheung et al., 2010)

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