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The specific mechanism may also be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis, thus promote angiogenesis of gastric mucosa and also interact with endoplasmic reticulum (ER) stress

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Interestingly, B12 decreased intracellular protein levels of IL-1B in the MOD group ( Figure 3F )

Editorial: targeting cardiac proteotoxicity