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Ca 2+ dependent potentiation of the nonselective cation channel TRPV4 is mediated by a C-terminal calmodulin binding site

Quality control of the reads was performed using in-house programs

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Following activation by redox signaling downstream of the MAPK signal pathway, the EpRE binding protein, also an oxidative stress-associated transcription factor, induces the dissociation of Nrf2 from Keap1 via redox modification and/or phosphorylation of Nrf2

Despite being frequently prescribed, MTXs severe multiple toxicities can occasionally limit its therapeutic potential