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However, lysosomal dysfunction compromises this clearance mechanism, leading to intraneuronal build-up and aggregation of -synuclein 49

Nat Rev Drug Discov 16:487511

This valuable and informative approach appears not to have been explored further in the context of PD models, although the recent report of a rat knockout model 34 should extend the opportunity to do so

Antioxidation Oxidative stress plays a central role in the development of PD, primarily through the excessive accumulation of free radicals and reactive oxygen species (ROS), thereby leading to neuronal damage and cell death ( The treatment of PD must emphasize on neuron survival and regeneration promotion after defending neurons against oxidative and inflammatory damage

Nitric oxide insufficiency, platelet activation, and arterial thrombosis

Cur-loaded LCS treatment in group 6 displayed a substantial rise in serum insulin levels and a reduction in insulin resistance, approaching normalization of values compared to controls