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Circulating GAD-reactive B cells that can differentiate into antibody producing cells have been also detected in the peripheral blood and bone marrow of patients with GAD-Ab associated neurological syndromes

Furthermore, additional receptors, such as TLR3, TLR4, and IFNAR1, play roles in initiating necroptosis, involving adapter molecules such as TIR domain-containing adapter molecule 1 (TICAM1, also known as TRIF) or RIHM-containing proteins Z-DNA binding protein 1 (ZBP1) [125, 126]

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