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doi: 10.4314/tjpr.v19i4.8 51 HaoYMiaoJLiuWPengLChenYZhongQ

After analyzing the effect of NR on the physiopathology and cellular mechanisms in the 3xTgAD/Pol mouse model (mice deficient in DNA repair that exacerbates the main characteristics of AD in humans) it was observed that supplementation with NR significantly increased the NAD + /NADH ratio, reduced neuroinflammation, apoptosis and damage to DNA, increased neurogenesis, restored synaptic plasticity, improved learning capacity and reversed memory deficits (Hou et al., 2017a)

Myristoylated FSP1 was recruited to the plasma membrane, and catalyzed the reduction of ubiquinone (CoQ 10 ) by using NAD(P)H to form ubiquinol that functioned as a radical-trapping antioxidant to terminate LPO and finally suppressed ferroptosis