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These complexities are further underscored by a recent report by Soriani et al., which suggests that CD38 can act as a decoy for extracellular cGAMP, a mediator of innate immunity

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Ultimately, determining how elevated GSSG outside the ER alters ER oxidative protein maturation will require compartment-specific monitoring and manipulation of the glutathione redox state

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Heatmap analysis revealed that Matrigel organoids exhibited broad transcriptional alterations following 5-FU exposure, whereas collagen organoids displayed a more restricted response in both mouse and human (Fig

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