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Deficiencies in four different drug elimination pathways have been explored in some detail, including glucuronidation (UGTs), acetylation (NATs), methylation (TPMT), and active transport (ABCG2)

Proposed mechanisms for all these increased risks included systemic low-grade inflammation, alterations in neuronal signaling, particularly dopamine and serotonin signaling pathways, and the influence of UPF additives on neurochemical regulation

Mizrachi et al., 2016)

Thus, the safety of remdesivir utilization in G6PD-deficient subpopulations needs to be elucidated and requires further research

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