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Neuroprotection research includes hippocampal ischemia-reperfusion models in rats (PMID 32558293) and investigations into the brain-gut axis as a candidate explanatory framework for the broad effects observed across tissue types (PMID 27138887)

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VPS35 gene mutations can lead to mitochondrial dysfunction and autophagy defects, preventing the timely clearance of aggregated proteins and thereby promoting Lewy body formation, followed by damage to dopaminergic (DA) neurons [18]