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In pathological settings, iron excess elevates the generation of oxygen free radicals, which subsequently exacerbates the oxidation and destruction of many cellular components [71]

The resulting soluble supernatant (crude extract) was used to determine G3PDH activity

The redox inactivation of SAL1 promotes the transport of PAP from chloroplasts (and mitochondria) via PAPST transporters to the nucleus where it inhibits exoribonuclease-mediated RNA metabolism (Estavillo et al., 2011

105 Optimal treatment and storage conditions are essential to prevent cellular deterioration and guarantee their viability for future scientific investigation

doi: 10.1016/j.neuroscience.2019.01.024

[PubMed: 16079258] 33
