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In pathological settings, iron excess elevates the generation of oxygen free radicals, which subsequently exacerbates the oxidation and destruction of many cellular components [71]

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The resulting soluble supernatant (crude extract) was used to determine G3PDH activity

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The redox inactivation of SAL1 promotes the transport of PAP from chloroplasts (and mitochondria) via PAPST transporters to the nucleus where it inhibits exoribonuclease-mediated RNA metabolism (Estavillo et al., 2011

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105 Optimal treatment and storage conditions are essential to prevent cellular deterioration and guarantee their viability for future scientific investigation

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doi: 10.1016/j.neuroscience.2019.01.024

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[PubMed: 16079258] 33

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