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We also proposed an alternative explanation for the pathogenic role of allicin in ischemic stroke by promoting protective genes and included a detailed potential discussion

Shabanzadeh, Sorensen, Jorgensen 2017b

Acute cerebral hypoxia caused by thrombosis during ischemia triggers inflammatory cascades, and immune activation can cause secondary damage within the penumbra of the ischemic infarct (Scheiermann et al., 2013)

The small molecule quinolinol MMRi62 has also demonstrated potential in overcoming gemcitabine resistance by promoting the proteasomal degradation of mutant p53 and the lysosomal degradation of NCOA4 and FTH1, ultimately inducing ferroptosis