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Description
Reciprocal regulation of acetyl-CoA carboxylase 1 and senescence in human fibroblasts involves oxidant mediated p38 MAPK activation
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proangiogenic factors (i.e., upregulated after wound healing even in the absence of new vessels) (for a review, see, i.e., [126,127,128])
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USA 93 , 84908495 (1996)
Moreover, co-incubation of in vitro generated dormant CSCs with E-pre-treated neutrophils, but not other immune cells resulted in an exit from the dormancy and renewed tumour formation
