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Interestingly, genetic impairment of NMDAR function specifically within cortico-limbic inhibitory interneurons is sufficient to cause schizophrenia phenotypes [191], which is consistent with the observation that NMDAR blockers can result in suppressed activation of inhibitory interneurons, resulting in dis-inhibition of excitatory neurons [174]
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coli, is a 20kDa protein containing 161 amino acid residues of the UCN3Human, 1-161 amino acids
The same low values were obtained in both control groups sacrificed either after 15 or 28 days
