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Controlling the role of BPC 157 likely involves its particular interaction with the major systems involved in wound healing, such as the NO-system (counteracted harmful events, arising from either NO-blockade or NOS-substrate application) (Sikiric et al., 2014) and the prostaglandins system (i.e., large extent of the counteracted NSAIDs toxicity (Sikiric et al., 2013) as well as counteracted developing and already formed adjuvant arthritis in rats (Sikiric et al., 1997c))

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RAS/MAPK signaling pathway and its importance in cancer Following the autophosphorylation of receptor tyrosine kinases, such as c-Met or many other receptor types, the activated receptor binds to the adaptor protein growth-factor-receptor-bound protein 2 (GRB2)

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