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PPAR/PGC1 signaling as a potential therapeutic target for mitochondrial biogenesis in neurodegenerative disorders

doi:10.1081/clt-200068842

GSH reacts with dithionitrobenzoic acid (DTNB) (Akerboom and Sies, 1981) and by reducing GSSG total GSH (GSH + GSSG) can be measured

The optimal therapeutic approach may depend on the specific clinical indication, individual patient factors, availability of different treatment options, and the regulatory framework within the practicing jurisdiction

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