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Using SPECT/CT imaging, we observed that MPN chassis localized specifically to areas containing atherosclerotic lesions, but only in LDLR / mice, which develop atherosclerosis under high-fat diet conditions (Fig

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Reduced use of cholesterol and sphingomyelins is sufficient to promote synapse formation As ncr-2(0) mutants exhibited higher levels of cholesterol and sphingomyelin in comparison with WT, we next explored whether the synaptic phenotypes observed in ncr-2(0) mutants were attributable to the elevated cholesterol and sphingomyelin levels

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