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To confirm that the induced cytotoxicity was indeed driven by ferroptosis, we employed Lip-1, a standard ferroptosis inhibitor that functions through its RTA activity [19]
PubMed Necas M
4d), which were enriched in pathways including ROS, OXPHOS, glycolysis, and DNA repair (Fig
A treatment regimen can be created once there has been a confirmed diagnosis
G., Maggi, V., Kok, J
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