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These agents mimic the biological action of the native incretin hormone GLP-1 but are structurally modified to resist degradation by the enzyme dipeptidyl peptidase 4 (DPP-4), thereby extending their half-life and duration of action (Zheng et al., 2024

Inhibition of TH by -methyltyrosine (-MT) alleviates WT or mutant -syn-induced dopaminergic neuron toxicity [120]

(2014) Oxidative stress and mitochondrial dysfunction in alzheimers disease