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In Metal Ions in Life Sciences Vol

Overexpression of schizophrenia susceptibility factor human complement C4a promotes excessive synaptic loss and behavioral changes in mice

Evidence from animal models and in vitro experiments provides substantial support for the contribution of ferroptosis to PD pathogenesis

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When the enzyme is allowed to react with a -glutamyl donor in the absence of an acceptor substrate, the -glutamyl group is transferred to another molecule of the -glutamyl donor, which results in the formation of a -glutamyl--glutamyl compound and/or a -glutamyl--glutamyl--glutamyl compound [89]

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