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These findings corroborate evidence of decreased Complex II protein levels and OXPHOS activity, which is a marker for mitochondrial FAD in muscle of patients with severe neuromuscular phenotype and novel variants in SLC25A32 [30,31]
C., Hunt J
See the full evidence review for a study-by-study breakdown
She adds, these symptoms are made worse by an enhanced production of IL-6 in response to pain
Wilbur DS, Pathare PM, Hamlin DK, Buhler KR, Vessella RL
The bloodbrain barrier: an overview: Structure, regulation, and clinical implications
