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During the development of nonalcoholic fatty liver disease (NAFLD), disorders of the carnitine pool are considered important drivers, in which the LCAC profile changes significantly with the progression of NAFLD and provides key clues for revealing the complex pathogenesis of NAFLD [209, 210]

These lesions may be clinically misdiagnosed as hemangiomas The classic SN (Fig

A 3D atlas of the dynamic and regional variation of pancreatic innervation in diabetes

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