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& Pollara, J
The proposed primary mechanism involves augmentation of hepatocyte growth factor (HGF) / c-Met receptor signaling, with downstream activation of MAPK, PI3K-Akt and the canonical synaptic-protein-synthesis machinery [2]
Thus, the antidepressant response is the result of durable changes in the brain that develop after transient NMDA receptor inhibition, not with sustained inhibition
Chan HP, et al
These results are shown graphically in Fig
We observed a significant increase (60%) in hexokinase activity in the GK rat liver, which remained higher than control levels even after exercise (Figure 4A)
