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In a monosodium iodoacetate-induced OA rat model, Methyltransferase-like 14 (METTL14) was confirmed to inhibit ferroptosis and ECM degradation in damaged chondrocytes by inhibiting m6A modification of GPX4 mRNA, a common RNA methylation, thereby protecting cartilage (Liu D

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This article examines that question through three complementary lenses: patient experience, mechanistic science, and clinical delivery

All data were collected and analyzed by research personnel blinded to participants group assignments except those involved in the intervention