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The authors would also like to thank Dr

Further, we introduce the research currently underway on other structural derivatives of NAC and acknowledge the continuum of efforts through pre-clinical and clinical research to facilitate further therapeutic development of NAC or its derivatives for rare diseases

Hypoxia, worsened by toxin-induced vascular dysfunction, further activates HIF-1 pathways that drive invasion and therapeutic resistance [114]

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Yang et al (47) demonstrated that reduced GPX4 expression may lead to the induction of ferroptosis, whereas increased GPX4 expression may inhibit ferroptosis

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