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Recent studies have demonstrated the cell deaths role in the homeostasis of the intestinal epithelium in IBD (137)

These results revealed that CopR functions as a regulator that negatively regulates copB gene and Cu 2+ serves as inducer of CopR to activate the ATPase CopB to export cupric ions under H 2 O 2 stress in L

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Additionally, supplements like N-acetylcysteine (NAC) can help restore glutathione levels

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