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Rapprich, S., Dingler, A
One interesting finding indicates that SFN administration successfully reduced endothelial cell death and eliminated TJ proteins, facilitating its role in preserving the BBB (91)
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Neurons and astrocytes both preferentially express the mitochondrially-located, kidney-type glutaminase (GLS) isoenzyme type 1 (GLS1), although there is also the liver-type GLS2 that localizes to the mitochondrion and nucleus (Cardona et al., 2001
Nuclear factor-erythroid 2-related factor 2 (Nrf2), a transcription factor that functions as a master regulator of cellular metabolism and anti-oxidant response [11], is also suppressed in patients with FA, in part due to FXN deficiency [12]
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