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The most pronounced changes were observed in the expression of -glutamate-cysteine ligase ( gshA ) and isocitrate dehydrogenase (icdA), which were upregulated by approximately 2-fold

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HOMOCYSTEINE AND METHYLMALONIC ACID As we have seen, in anemia due to B12 deficiency, both homocysteine and methylmalonic acid are elevated, while in B9 deficiency (also a cause of megaloblastic anemia), only homocysteine is found in plasma levels above normal

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Preventing Chronic Disease [Electronic Resource], 17, E106

Notably, depletion (rather than pharmacologic inhibition) of PRMT1 significantly suppressed fibroblast proliferation, linking intracellular arginine methylation to fibrotic remodeling [90]