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DLK-1, SEK-3 and PMK-3 are required for the life extension induced by mitochondrial bioenergetic disruption in C

Lysine Acetyltransferase 6A (KAT6A) decreased in old MSCs, coinciding with reduced proliferation, colony formation, and osteogenesis of MSCs by regulating antioxidant gene Nrf2 to inhibit ROS accumulation [111]

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[6] [9] [5] While previously known as a common cause of death in pregnancy, with proper treatment this is now very rare

ChIP assay showed REX1 directly bound to MKK3, an upstream regulator of p38 MAPK, to exert its effects on MSC stemness [49] (Fig

10.1212/WNL.0000000000002461 Neurology 226 PanJ.ChangQ.WangX.SonY.ZhangZ.ChenG.et al (2010)