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Mutation of p53 and Ki-ras gene in human fetal lung fibroblast cells in vitro by sterigmatocystin

[Google Scholar] Kurniawan H., Franchina D

Collectively, these investigations suggested the plausibility of modulating the rate of drug release as well as specifically targeting different GSTs by modifying the sulfonamide moiety 128

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We performed a side-by-side comparison of all ER-targeted constructs with all cell surface-targeted constructs (Fig