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10.1176/appi.ajp.2015.14101358 23 BolzS

These results suggest that the putatively vesicular copper released from neurons following NMDAR stimulation (Schlief et al., 2005) may act on NMDARs themselves to rein in excessive excitation, and that additional copper release can protect neurons from excitotoxic cell death

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up to 24% in obesity trials) Constipation (6-17% of patients, dose-dependent) Abdominal pain and dyspepsia (6-11% of patients) These gastrointestinal effects are typically most pronounced during dose escalation and often diminish over time as patients develop tolerance