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Final evidence for a causal role of elevated levels of cytosolic or extracellular self-DNA as a driver of systemic inflammation in severe COVID-19 remains to be provided, and it will be difficult to separate the TLR-mediated pro-inflammatory effects of CoV-2 S from pro-inflammatory effects that are as suggested by the present work caused by self-DNA release due to CoV-2 S-mediated damage to genomic DNA and the nuclear envelope

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About this article Cite this article Chen, J.J., Thiyagarajah, M., Song, J

When hypermethioninemia results from inherited mutations in at least three genes associated with methionine metabolism it is referred to as primary hypermethioninemia