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The alterations in glutamine metabolism in tumor cells are an important outcome of the changes in the energy metabolism of tumor cells

The structural peculiarities of CD44 are thus very important towards translational medicine

The authors proposed involvement of the TGF-beta-1/Smad axis

FIGURE 1 3.1.1.3 Cancer metastasis and ROS Excessive level of ROS leads to metastasis via the stimulation of mitogen-activated protein kinase (MAPK), and phosphoinositide-3-kinase regulatory subunit/AKT serine/threonine kinases/mechanistic target of rapamycin kinase (PI2K/Akt/mTOR) signaling pathways that directs the activation of downstream metalloproteinase 9 (MMP9), metalloproteinase 2 (MMP2), SNAIL enzymes which initiate epithelial-mesenchymal transition (EMT) to metastasis ( 3.1.1.4 Pro-angiogenic role of ROS Activation of angiogenesis through ROS via vascular endothelial growth factor (VEGF) independent and VEGF dependent pathways are shown in Figure 2

An activation of caspases was also detected (Fig

FRET ratio ( Em YFP/ Em CFP) was monitored with specified intervals