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The mice lacking microglial TLR-4 were protected from NEC associated brain injury

The loss of dopaminergic neurons means these cells are no longer available to regulate the concentration or spatial distribution of dopamine via DAT-mediated uptake, potentially exposing immune cells to aberrant dopamine concentrations that could drive inflammatory activity

doi: 10.1093/rheumatology/kep080

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Antimicrobial prophylaxis should be procedure- and risk-specific rather than administered solely because mesh is implanted