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KRAS mutations reprogram cancer metabolism, resulting in increased glucose uptake and glycolysis [7], altered glutamine/glutamate metabolism [8, 9], abnormal mitochondrial activity and redox equilibrium [1012], which are critical for the increased energetic, biosynthetic, and redox requirements of tumor cells and therefore required for KRAS- induced tumorigenicity [13, 14]

But some plant GSTs show activity on ITCs in vitro 37,38,39 , and exogenous application of AITC upregulates transcript levels of GST s and depletes glutathione 17,40,41

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also examined the effect of Epitalon on melatonin release in isolated perifused pineal glands from both young and aged rats

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