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Based on domain architecture comparison with HNF4 58,59 , we engineered an NHR-49 truncation (295422) to disrupt ligand binding by removing a majority of the ligand binding domain, while retaining its analogous N-terminal, DNA-binding, hinge, and C-terminal domains (Fig

Bacterial bile salt hydrolase in host metabolism: potential for influencing gastrointestinal microbehost crosstalk

Anafarta, K., Bedk, Y., Uluoglu, O., Aydos, K

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This restorative and soothing effect may be due to the anti-inflammatory and moisturizing properties of the various actives

[22] Accumulation of APOCIII on HDL is important for the maintenance of plasma triglyceride homeostasis since it prevents excessive amount of APOCIII on VLDL and other triglyceride rich lipoproteins, thus preventing APOCIII-mediated inhibition of LpL and the subsequent hydrolysis of plasma triglycerides